The physiological mechanisms of the hot flash efferent responses, particularly in the cutaneous circulation, are not completely understood. 4.
cutaneous vasodilation Cutaneous Skin blood flow and perceptual measures follow a similar time course as menthol appearance/clearance.
Mechanisms This review focuses on the neural and local mechanisms that have been demonstrated to effect cutaneous vasodilation and vasoconstriction in response to heat and cold stress in vivo in humans. Menthol induces cutaneous vasodilation, however the underlying mechanisms are
Mechanisms During heat stress, increases in blood flow in nonglabrous skin in humans are mediated through active vasodilation by an unknown neurotransmitter mechanism.
Mechanisms and time course of menthol-induced cutaneous Cutaneous Almost 70 years ago the human cutaneous active vasodilator system was first described; however, its mechanisms remain unclear today. The mechanisms by which sympathetic nerves mediate cutaneous active vasodilation during whole body heating and cutaneous vasoconstriction during whole body The physiological mechanisms of the hot flash efferent responses, particularly in the cutaneous circulation, are not completely understood. Current concepts for the mechanisms that effect local cutaneous vascular responses to local skin warming and cooling are examined, including the roles of temperature sensitive The physiological mechanisms of the hot flash efferent responses, particularly in the cutaneous circulation, are not completely
Cutaneous Active Vasodilation in Humans Is Mediated by Kellogg et al.
Mechanisms Mechanisms and time course of menthol-induced Though the hypercapnea caused vasodilation is well known, the underlying physiological mechanism remains still obscure. Abstract During heat stress, increases in blood flow in nonglabrous skin in humans are mediated through active vasodilation by an unknown neurotransmitter mechanism. The mechanisms by which sympathetic nerves mediate cutaneous active vasodilation during whole body heating and cutaneous vasoconstriction during whole body To investigate this mechanism, a three-part study was To test the hypothesis that baroreceptor though the precise mechanisms remain to be eluci-unloading during dynamic exercise limits cutaneous dated, it is clear that this system plays a vital role in vasodilation, several investigators had subjects per- To investigate this With regard to cutaneous vasoconstrictor mechanisms, a classic study from Zelis and colleagues showed augmented vasoconstriction in the skin at the onset of and Increases in skin blood flow during postmenopausal hot flashes are neurally mediated primarily through BTX-sensitive nerves, presumably sympathetic cholinergic. To investigate this mechanism, a three part study was performed to determine the following: (1) is muscarinic receptor activation necessary for active cutaneous vasodilation?
Mechanisms This review focuses on the neural and local mechanisms that
Cutaneous Thermoregulation during Exercise in the Heat - University of in response to metabolic or environmental disturbances to heat balance, the autonomic nervous system initiates cutaneous vasodilation and eccrine sweating to facilitate higher rates of dry (primarily convection and radiation) and evaporative transfer from the body surface; however, absolute heat losses are ultimately governed by the properties of mechanism of cutaneous active vasodilation.
Cutaneous Vasodilator and Vasoconstrictor Mechanisms in Search 18 grants from Christopher Minson
Cutaneous Vasodilator and Vasoconstrictor Mechanisms in In humans, vasoactive intestinal peptide (VIP) may play a role in reflex cutaneous vasodilation during body heating. Objective: Menopausal hot flashes can seriously disrupt the lives of symptomatic women.
Mechanisms We tested the hypothesis that the nitric oxide (NO)-dependent contribution to active vasodilation is enhanced in the skin of subjects with cystic fibrosis (CF), compensating for sparse levels of VIP. Materials and Methods Topical menthol is detectable in the skin within Menthol induces cutaneous vasodilation in the skin through multiple vasodilator pathways, including NO, EDHF, and sensory nerves. In this study, we investigated the possible mediators of skin vasodilation that is evoked by bathing with the CO2-water in experimental animals. The aim of this study was to examine the mechanisms of increases in skin blood flow during the postmenopausal hot flash in symptomatic women.
In vivo mechanisms of cutaneous vasodilation and Asc and Asc+A-I augmented cutaneous vasodilation in Cutaneous vasodilation and sudomotor activity are controlled by a sympathetic cholinergic active vasodilator system that is hypothesized to operate through a co-transmission
cutaneous vasodilation During heat stress, increases in blood flow in nonglabrous skin in humans are mediated through active vasodilation by an unknown neurotransmitter mechanism. https://journals.physiology.org/doi/full/10.1152/japplphysiol.01071.2005
In vivo mechanisms of cutaneous vasodilation and Topical menthol is detectable in the skin within 30min and is cleared by 60min.
Mechanisms mechanisms Local thermal control of the human cutaneous circulation cutaneous vasodilation Mechanisms of Cutaneous Vasodilation in Humans Mechanisms of Cutaneous Active Vasodilation - Dean Kellogg Cutaneous vasodilation was attenuated in essential hypertensive skin (HTN: 353 vs. AMN: 424 %CVCmax, p<0.05).
In vivo mechanisms of cutaneous vasodilation and In vivo mechanisms of cutaneous vasodilation and vasoconstriction in humans during thermoregulatory challenges.